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human melanoma cell line sk mel 2  (Procell Inc)

 
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    Procell Inc human melanoma cell line sk mel 2
    (A) Six predicted cER genes were <t>investigated</t> <t>in</t> <t>SK-mel-2</t> cell line ( n = 4). (B) Six predicted cER genes were investigated in Caki-1 cell line ( n = 4). Statistical analysis was performed between si-NC and si-Genes. P -values are calculated by One-way analysis of variance followed by Dunnett’s corrections and indicated by star symbols, *, P < 0.05; **, P < 0.01; ***, P < 0.001; ****, P < 0.0001; ns, P > 0.05.
    Human Melanoma Cell Line Sk Mel 2, supplied by Procell Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/human+melanoma+cell+line+sk+mel+2/pmc13138745-403-1-34?v=Procell+Inc
    Average 86 stars, based on 1 article reviews
    human melanoma cell line sk mel 2 - by Bioz Stars, 2026-07
    86/100 stars

    Images

    1) Product Images from "CASER: A semi-supervised model with multi-omics data integration prioritizes cancer-associated epigenetic regulator genes"

    Article Title: CASER: A semi-supervised model with multi-omics data integration prioritizes cancer-associated epigenetic regulator genes

    Journal: PLOS Computational Biology

    doi: 10.1371/journal.pcbi.1014253

    (A) Six predicted cER genes were investigated in SK-mel-2 cell line ( n = 4). (B) Six predicted cER genes were investigated in Caki-1 cell line ( n = 4). Statistical analysis was performed between si-NC and si-Genes. P -values are calculated by One-way analysis of variance followed by Dunnett’s corrections and indicated by star symbols, *, P < 0.05; **, P < 0.01; ***, P < 0.001; ****, P < 0.0001; ns, P > 0.05.
    Figure Legend Snippet: (A) Six predicted cER genes were investigated in SK-mel-2 cell line ( n = 4). (B) Six predicted cER genes were investigated in Caki-1 cell line ( n = 4). Statistical analysis was performed between si-NC and si-Genes. P -values are calculated by One-way analysis of variance followed by Dunnett’s corrections and indicated by star symbols, *, P < 0.05; **, P < 0.01; ***, P < 0.001; ****, P < 0.0001; ns, P > 0.05.

    Techniques Used:



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    Image Search Results


    (A) Six predicted cER genes were investigated in SK-mel-2 cell line ( n = 4). (B) Six predicted cER genes were investigated in Caki-1 cell line ( n = 4). Statistical analysis was performed between si-NC and si-Genes. P -values are calculated by One-way analysis of variance followed by Dunnett’s corrections and indicated by star symbols, *, P < 0.05; **, P < 0.01; ***, P < 0.001; ****, P < 0.0001; ns, P > 0.05.

    Journal: PLOS Computational Biology

    Article Title: CASER: A semi-supervised model with multi-omics data integration prioritizes cancer-associated epigenetic regulator genes

    doi: 10.1371/journal.pcbi.1014253

    Figure Lengend Snippet: (A) Six predicted cER genes were investigated in SK-mel-2 cell line ( n = 4). (B) Six predicted cER genes were investigated in Caki-1 cell line ( n = 4). Statistical analysis was performed between si-NC and si-Genes. P -values are calculated by One-way analysis of variance followed by Dunnett’s corrections and indicated by star symbols, *, P < 0.05; **, P < 0.01; ***, P < 0.001; ****, P < 0.0001; ns, P > 0.05.

    Article Snippet: The human melanoma cell line SK-mel-2, clear cell renal cell carcinoma cell line Caki-1, breast cancer cell line MDA-MB-231, and prostate cancer cell line LNCaP, along with their corresponding complete median, were purchased from Procell Life Science & Technology Co., Ltd. (Wuhan, China).

    Techniques:

    Journal: bioRxiv

    Article Title: Human melanoma cell lines that possess wild-type BRAF alleles but are dependent on ERBB4 and ERBB2

    doi: 10.1101/2024.08.22.609260

    Figure Lengend Snippet:

    Article Snippet: The IPC-298 [ ], MEL-JUSO [ ], MeWo [ ], and SK-MEL-2 [ ] human melanoma cell lines were obtained from ATCC [ ] (Manassas, Virginia, USA) and DSMZ [ ] (Braunschweig, Germany).

    Techniques: Dominant Negative Mutation, Mutagenesis